A new UCLA Health study in mice has identified a new potential trigger for pulmonary fibrosis: lipid dysregulation (a fat imbalance) within alveolar macrophages. These are the immune cells that serve as the first line of defense against pathogens and tissue damage. Under normal conditions, they are essential for the maintenance and repair of the lung’s airspaces. However, when these cells suffer from a lipid imbalance, they lose their protective qualities, malfunction and begin actively promoting disease.
If you liked the article, do not forget to share it with your friends. Follow us on Google News too, click on the star and choose us from your favorites.
If you want to read more Like this articles, you can visit our Science category.
